As a result of this study’s findings, it is now of interest to determine if viruses implicated in ME/CFS [11], or the proteins they create, have the capacity to catalyze fibrinaloid microclot formation, as we showed that the S1 subunit of the SARS-CoV-2 spike protein is capable of doing in vitro in Long COVID/PASC PPP [49]. The gene discussed is PSMD1; the disease is myalgic encephalomeyelitis/chronic fatigue syndrome.