Upon infection with Jan-E EHV-1, we observed a progressive decrease in the amount of LC3B protein present in the form of aggregates, partially colocalizing with mitochondria at 2 and h p.i. and completely colocalizing at 48 h p.i. (Figure 9C) Infection with the Rac-H EHV-1 resulted in increased amounts of LC3B protein during infection, with a particular increase at 48 h p.i. (Figure 9D). The gene discussed is MAP1LC3B; the disease is infection.