Recently, Si et al. (2020) found that silybin treatment elevates mitochondrial fusion through an increase in the expression of mitochondrial fusion-associated proteins (optic atrophy 1, mitofusin 1 and mitofusin 2) and down-regulation of the expression of the mitochondrial fission-associated protein (dynamin-related protein 1 (DRP1)) in MDA-MB-231 breast cancer cells, which consequently impedes cellular migration [124]. This evidence concerns the gene DNM1L and breast cancer.