Moreover, a reduction in polymerisation and aggregation of αSyn was demonstrated in hSOD1G93A mice treated with bee venom, a traditional medicine for inflammatory diseases used to rescue the impairment of the ubiquitin-proteasome system in ALS, further strengthening the hypothesis surrounding the relationship between αSyn and SOD1 [138,139]. The gene discussed is SOD1; the disease is amyotrophic lateral sclerosis.