CFTR and cystic fibrosis: In this context, the possibility of PNA-dependent modulation of miRNAs involved in the regulation of CFTR expression is of great impact in the case this approach will be demonstrated to be efficient in further increasing the activity of drugs already employed for personalized therapy of CF, such as, for instance, read-through molecules [62,63,64] and CFTR correctors [65,66,67].