Over 30 mutations have been found to correlate with ALS, particularly its familial form, in genes, such as superoxide dismutase 1 (SOD1), transactive response (TAR)-DNA binding protein (TARDBP, previously called TDP43), angiogenin (ANG), fused in sarcoma RNA binding protein (FUS), and chromosome 9 open reading frame 72 (C9orf72) [3,4]. This evidence concerns the gene TARDBP and amyotrophic lateral sclerosis.