Since the damage induced during the development of the brain cortex results in severe sensory and motor impairments in infants with HIE, and considering the therapeutic potential of GH on brain repair after neural injury or stroke in both clinical [31,32,33,34,35] and preclinical [36,37,38,39,40,41] studies, it is pertinent to further investigate the possible protective and restorative actions of GH in the brain cortex after prenatal HI injury. The gene discussed is GH1; the disease is stroke disorder.