Two studies included in the review (n = 2/27, 7%, n = 27) seemed to suggest that LL-37 increases and sustains inflammation in the lungs over the course of asthma by acting as a chemotactic factor for eosinophils and neutrophils [48] and boosting the release of proinflammatory cytokines [63]. The gene discussed is CAMP; the disease is asthma.