In addition, consistent with “omics technologies” [54], our results provide evidence of the heterogeneity of IL-3Rα response in different TNBCs (the ability to induce the expression of EMT marker was tumour-specific), as well as on the ability of IL-3Rα to activate common pathways (the induction of the epithelial-to-endothelial switch was detected in all the cell lines tested). The gene discussed is IL3RA; the disease is neoplasm.