ICAM1 and Sepsis: Studies investigating the molecular mechanisms of neutrophils’ recruitment into the kidney discovered that two β2-integrins, macrophage-1 antigen (Mac-1) and lymphocyte function-associated antigen 1 (LFA-1); two adhesion molecules, ICAM-1 and vascular cell adhesion molecule 1 (VCAM-1); as well as E-selectin and P-selectin are involved in neutrophil recruitment into the kidney after the induction of sepsis.