CXCL10 and infection: In summary, we were able to assign monocytes from both infection models to recently suggested subgroups with a more proinflammatory and activated phenotype in the L. monocytogenes model (nMO; dcMO; Cxcl10+ and Saa3+ monocytes) and a scarcely activated phenotype in the parasite model characterizing Ly6Chi to Ly6Clo converting monocytes.