TP53 and cervical carcinoma: Part of the mechanism involved in dexamethasone-mediated proliferation and survival of cancer cells from solid tumors involves glucocorticoid receptor-mediated activation or suppression of target genes, modulation of the P53-dependent miR-145 expression in HPV-positive cervical cancer cells, and activation of HPV through the responsiveness of the upstream regulatory region (URR) of the virus [194,195].