IL6 and COVID-19: Considering this pathogenetic mechanism, several recent studies reported an increased level of proinflammatory cytokines and chemokines such as tumor necrosis factor-α (TNF-α) and interleukin-6 (IL-6) in COVID-19 patients [16,17] suggesting that this cytokine storm might have a critical role in the evolution of SARS-CoV-2 infection [18].