BAP1 and Miyoshi myopathy: Among them, the germline mutations of the tumor suppressor BRCA1 Associated Protein 1 (BAP1) and the exposure to Simian Virus 40 (SV40) are considered to be the main contributors [238,239], although there is no direct evidence that the presence of BAP1 mutations or SV40 infection modulates autophagy to regulate the development of MM.