IL22 and dry eye syndrome: Additionally, Qiao et al. inhibited miR-6731 in IL-22-stimulated HaCaT cells, showing an increased proliferative activity of the IL-22-induced KCs, while protein expression levels of therapeutic targets of PsO, including TNFα, IL-23, HLA-C as well as inflammatory molecules such as NF-κB and the PSORS1 locus were significantly overexpressed.