AKT1 and type 2 diabetes mellitus: The experiments of Wang et al. [41] and Wang et al. [42] reported that OA decreased the protein expression of glucose-6-phosphatase (G-6-Pase) and phosphoenolpyruvate carboxykinase (PEPCK) and increased the AMP-activated protein kinase (AMPK) and Akt phosphorylation in livers of type 2 diabetic mice.