The pathophysiology of BD is not completely understood, although there is increasing evidence regarding the role of inflammation [5,6,7,8], as supported by a recent systematic review where the levels of interleukin-8 (IL-8), monocyte-chemoattractant protein-1 (MCP-1), eotaxin-1 and interferon-γ-induced protein 10 (IP-10), which are chemokines associated with inflammation, were found to be higher in BD patients than in healthy controls [9]. This evidence concerns the gene CCL11 and Behcet disease.