METTL3 exerts its oncogenic role in lung cancer by enhancing the translation of epidermal growth factor receptor (EGFR), transcriptional co-activator with PDZ-binding motif (TAZ) and DNA methyltransferase 3A (DNMT3A) in a methyltransferase- or m6A binding protein-independent manner by recruiting the translation initiation factor eIF3h to METTL3 bound transcripts [30]. This evidence concerns the gene EGFR and lung cancer.