Additionally, a cationic AMP GW-H1 exerts highly selective cytotoxicity against AGS gastric cancer cell lines via both apoptosis and autophagy in the early stage, and the caspase-dependent apoptosis was further enhanced by inhibition of autophagy at the final stage, with beclin-1 serving as a promising target for inhibiting autophagy to sensitize GW-H1 therapy for gastric cancer [43]. Here, BECN1 is linked to gastric cancer.