(2) The ARB treatment attenuated the Fas/FasL-mediated apoptotic protein levels (Fas Ligand, Fas death receptors, FADD, active caspase-8), and mitochondria-mediated apoptotic protein levels (Bax, cytochrome c, active caspase-9, and active caspase-3) in hypertension. This evidence concerns the gene FADD and hypertensive disorder.