CCL2 and hyperuricemia: This relationship between SUA and adverse lipid profile could be owed to hyperuricemia promoting LDL-C oxygenation, lipid peroxidation, vascular smooth cell proliferation, platelet adhesiveness, and synthesis of proinflammatory factors like monocyte chemoattractant protein-1, interleukin-lβ, interleukin-6, and tumor necrosis factor-alpha (TNF-α), thus promoting atherosclerotic arterial occlusion [9-12].