There is substantial evidence that infection by coronaviruses, including MERS-CoV and SARS-CoV-2, directs the immune response along the Th17 pathway [28–32], and IL-17 may represent one of the key mediators of tissue damage, acute respiratory distress syndrome (ARDS), and cytokine storm in such viral infections [32–39]. Here, IL17A is linked to acute respiratory distress syndrome.