Mechanism research has been gradually deepened to the level of cell and molecular biology [24] and found that the SOD activity of rats with fatty livers caused by CCl4 combined with a high-fat and low-protein diet decreased significantly and reported that compound preparation of TCM could significantly inhibit the NF-κB pathway in the liver and abdominal aorta of type 2 diabetic rats with NAFLD. Here, NFKB1 is linked to metabolic dysfunction-associated steatotic liver disease.