In addition, levels of interferon (IFN) γ, IFN-γ-induced protein 10 (IP-10), interleukin (IL) 8, IL-10, IL-17A, tumor necrosis factor (TNF) α, platelet-derived growth factor (PDGF), and soluble intercellular adhesion molecule 1 (sICAM-1) seem all to be altered in GCA patients as compared to controls. This evidence concerns the gene CXCL10 and temporal arteritis.