CALR and neoplasm: The idea is that a given agent (e.g., anthracyclines chemotherapy) or process (e.g., photodynamic therapy or radiotherapy) promotes local tumour cell death concomitant with the release of damage‐associated molecular patterns (DAMPs), such as ATP, HSP70 and HMGB1,117 together with endoplasmic reticulum stress that promotes externalization of eat‐me signals, such as calreticulin (CRT).118