In diabetic cardiomyopathy, high glucose induces the expression of myocardial infarction-associated transcript (MIAT) and upregulates the death-associated protein kinase 2 (DAPK2) expression by sponging miR-22-3p, which consequently leads to cardiomyocyte apoptosis (67). Here, DAPK2 is linked to diabetic cardiomyopathy.