The treatment of UC with HQT mainly acts on biological processes such as cancer pathway, MAPK signaling pathway, TNF signaling pathway, insulin resistance, JAK-STAT pathway, Th17 cell differentiation, NF-kB, and other signaling pathways, involving biological processes such as endotoxin response, regulation of apoptosis signaling pathway, regulation of small molecule metabolic process, reactive oxygen metabolic process, and negative regulation of cell proliferation [27]. This evidence concerns the gene NFKB1 and cancer.