Next, using a Drosophila model of malignant tumor, wherein the activated oncogene Raf (RafGOF) cooperates with loss-of-function mutations in the conserved tumor suppressor scribble (scrib–/–) (28), we evaluated the effect of oral administration of the CCP–Zn on tumor progression in Drosophila and determined the relevant mechanism. This evidence concerns the gene SCRIB and neoplasm.