Of interest, STR is coexpressed with bitter taste receptor T2Rs in solitary chemosensory cells, rare chemosensing epithelial cells, of sinonasal cavity of CRS patients and suppresses T2R‐mediated release of antimicrobial peptides from the surrounding epithelial cells during sinonasal bacterial infection,75, 76 implicating STR in the inhibition of immune responses. Here, STATH is linked to congenital rubella syndrome.