MYC and breast neoplasm: This concordance can also be observed at the pathway level: two of the three pathways that were upregulated in breast tumours based on DiffRAC estimates also appear to be enriched among mRNAs that are stabilized in MDA-LM2 compared to MDA-MB-231 cell lines (MYC targets and mTORC1 signaling, Fig. 2h; example genes are shown in Fig. 2i), supporting a role of mRNA stability in deregulation of these key pathways.