By contrast, INHBA, coding for the inhibin beta A subunit of other TGF-β superfamily members, including the homodimer Activin A, the heterodimer activin AB or their antagonist Inhibin A (a heterodimer of INHBA and an α subunit)43, showed the highest pseudotime autocorrelation and very specific association with Highly infiltrative tumor clusters (TC12–15) (Fig. 6a, b). This evidence concerns the gene TGFB1 and neoplasm.