The chaperone HSPA5 is involved in ferroptosis (Zhu et al., 2017) and contributes to an iron‐dependent non‐apoptotic cell death that stabilizes GPx4 and indirectly attenuates lipid peroxidation in glioma cells (Chen et al., 2019); therefore, we excluded HSPA5 from further study. This evidence concerns the gene GPX4 and glioma.