HNRNPUL2 and autism: Although MARK2, SCAF1 and HNRNPUL2 were not identified as archetypal (potentially suggesting divergence from well-known autism risk mechanisms), a search for functional enrichment of this interstitial region between A4 and A5 found that their roles in developmental risk may be most relevant at the cell–cell junction, particularly as it relates to migration (see Fig. 7d).