As shown in Figure 1I–L, Gstz1 deletion (Gstz1−/−) mice exhibited a higher metastatic ability and tumour burden than wild‐type (WT) mice, based on increased lung nodules (Figure 1J), larger and more numerous microscopic pulmonary lesions (Figure 1K,L), higher liver‐to‐body weight (LW/BW) ratios and higher serum aspartate aminotransferase (AST) activity (Figure S1L,M). The gene discussed is GSTZ1; the disease is neoplasm.