We also observed that Mettl3 or Mettl14 overexpression significantly attenuated DLL-enhanced Stat1 and Irf1 in 3LL tumor cells (Figures 6(a) and 6(b)), and MeRIP-qPCR results showed that Mettl3 or Mettl14 overexpression significantly enhanced the m6A methylation levels of Stat1 and Irf1 that were reduced by DLL treatment (Figure 6(c)). The gene discussed is IRF1; the disease is neoplasm.