To characterize the kinetics of the anti-apoptotic effect of IFNγ on CML cells, the proportion of active caspase-3/7 positive cells was analyzed in JURL-MK1 and K562 cells left untreated or treated with imatinib and/or IFNγ with fluorescence live cell microscopy (Figure 2). This evidence concerns the gene CASP3 and chronic myelogenous leukemia, BCR-ABL1 positive.