Transcripts for IFNG itself were scarce in all samples and often undetectable, preventing comparison of their abundance between tumor types; however, transcripts for IFNGR1 and IFNGR2 were readily detectable and similar across all tumor types (Figures S4G and S4H) indicating that decreased cytokine, rather than sensitivity to it, explains the suppressed IFN-γ response in SDHB/SDHD mutant tumors. The gene discussed is IFNGR2; the disease is neoplasm.