In addition, our human cellular sPD and fPD models as well as the Pink1 mouse model exhibit shortening of PC but no loss of cilia, which is similar to the phenotype observed in several mutations of the intraflagellar transport machinery, such as WDR35/IFT121 or WDR19/IFT144 mutants65–67. This evidence concerns the gene PINK1 and Platelet storage pool disease.