In addition, both the Cav1.2 pore‐forming Cacna1c and the regulatory beta‐subunit Cacnb2 are BD risk genes, and impaired cellular calcium homeostasis is a hallmark of BD (Harrison et al, 2019).This led us to hypothesize that Cav1.2 activity could be downstream of miR‐499‐5p in the control of dendritogenesis. The gene discussed is CACNB2; the disease is Behcet disease.