The results showed that the high-risk group was significantly enriched in functions related to the positive regulation of migration involved in sprouting angiogenesis, the phosphoinositide 3-kinase (PI3K)-AKT-mammalian target of rapamycin (mTOR) signalling pathway, basal cell carcinoma, extracellular matrix (ECM) receptor interaction, and angiogenesis, whereas the low-risk group was significantly enriched in apoptosis and the p53 signalling pathway. The gene discussed is AKT1; the disease is basal cell carcinoma.