At the same time, we also noticed that α-GA could indeed improve the apoptosis of liver tissue in NAFLD mice, accompanied by an increase in the level of Bcl-2 protein (p < 0.01) and a significant decrease in the level of Bax and cleaved-Caspase 3 protein (p < 0.01) (Figures 3B, C). This evidence concerns the gene BAX and metabolic dysfunction-associated steatotic liver disease.