The cytoplasmic hnRNP A1 destabilizes mRNA of cIAP1 in UV-irradiated cells (18), suppresses internal ribosome entry site (IRES)–mediated translation of X chromosome-linked IAP (XIAP) and B-cell lymphoma-extra large (Bcl-xL) during hypertonic shock (19), promotes nuclear export of XIAP and Bcl-xL mRNAs in FGF-2 stimulated cells (17), and drives translation of human rhinovirus RNA for efficient infection (20). This evidence concerns the gene BCL2L1 and infection.