Investigating a possible immunological marker of clinical response to baricitinib in RA patients, we longitudinally analyzed the frequencies and the STAT phosphorylation status of four selected immune cell types, i.e., monocytes, CD4 conventional T cells (Tconv), regulatory T cells (Treg), and CD8 T cells, identified according to the gating strategy shown in Supplementary Figure 1A. This evidence concerns the gene SOAT1 and rheumatoid arthritis.