Notably, mutations in DNA mismatch repair (MMR) genes were more frequent in STING‐high SCLC, whereas no mutations in these genes were detected in STING‐low SCLC tumors (Figure 4C), further confirming the inactivation of MMR pathway in STING‐high SCLCs (Figure 4B). This evidence concerns the gene STING1 and small cell lung carcinoma.