APOA1 and neuronal ceroid lipofuscinosis: This research could also be stimulated by the evidence that a deficiency of palmitoyl protein thio-esterase 1 (Ppt1), which causes an infantile form of neuronal ceroid lipofuscinosis (INCL), is associated in mice with increased ecto-F1-ATPase expression and apoA-I uptake in neurons, while serum cholesterol and apoA-I concentrations were decreased.