The pathogenesis of ADPKD involves excessive cell proliferation and the fluid secretion of the cyst-lining epithelium, which has been associated with the aberrant activation of cyclic adenosine monophosphate (cAMP), mTOR (mammalian target of rapamycin), and other pro-proliferative signaling pathways [5,6]. This evidence concerns the gene MTOR and autosomal dominant polycystic kidney disease.