Nevertheless, Skevaki et al., demonstrated that rhinovirus infection induces bFGF release by airway epithelium, and it stimulates stroma cell proliferation, contributing to airway remodeling in asthma, and Mehta et al., identified three molecules that contribute to fibrosis and lung tissue remodeling—LIGHT, IL-1β, and TGF-β—which are induced by Rhinovirus [10,29]. The gene discussed is FGF2; the disease is asthma.