These results stay in agreement with our previous study [26,27,28] and the literature, as it has been reported that infections caused by rhinoviruses are associated with an increase in airway remodeling mediators, e.g., TGF-α, TIMP-2, HGF, MMP-10, LIGHT, IL-1β, GF-1, PDGF, endothelin, and TGF-β [3,10,29]. The gene discussed is IL1B; the disease is infection.