The levels of DDX5 mRNA and protein were shown to be significantly increased in cervical cancer cell lines (CaSki, HeLa HPV18-positive, SiHa HPV16-positive and C-33A HPV-negative) compared to the HaCaT cell line of human keratinocytes; DDX5 overexpression greatly improved the migration capacity of CaSki cells through the activation of TGF1-β pathway, with an increase in epithelial–mesenchymal transition markers and alterations of cell morphology [83]. Here, DDX5 is linked to cervical carcinoma.