Using an oxygen-induced retinopathy mouse model, Kim et al. (2016) examined the possible effects of amniotic membrane-derived Mesenchymal stem cells (AMSC) on retinal neovascularization by characterizing TGF-β1 under pathological conditions and showed that intraperitoneally injected of human AMSC migrated into injured retina tissue and suppressed neovascularization through the release of ocular over-expression of TGF-β1 [267]. The gene discussed is TGFB1; the disease is retinal disorder.