Proteomic analyses of tumours induced by Fgfr2 variants demonstrated consistent expression and phosphorylation of FGFR2 variants along with downstream signalling activities, which were distinct from the phosphoproteome of FGFR2-independent K14-cre;Brca1F/F;Trp53F/F;(Mdr1a/b−/−) tumours (Extended Data Fig. 9a–c). The gene discussed is KRT14; the disease is neoplasm.